Filtyp
Sprickbildning i puts på isolering har inträffat i ökande omfattning på senare tid. 1997 togs problemställningen upp vid Byggnadsmaterial, Lunds Tekniska Högskola. Genom brottmekansika beräkningar, renodlade laboratorieförsök samt studier på provväggar och analys av skadefall i praktiken har olika inverkande faktorer klarlagts. I denna artikel sammanfattas hittills framkomna resultat och hypoteser
Purpose To explore the association of baseline trabecular bone structure with incident tibiofemoral (TF) osteoarthritis (OA) and with increase in joint space narrowing (JSN) score. Methods The Multicenter Osteoarthritis Study (MOST) includes subjects with or at risk for knee OA. Knee radiographs were scored for Kellgren–Lawrence (KL) grade and JSN at baseline, 30, 60 and 84 months. Knees (KL ≤ 1)
Current sources of platelets for transfusion are insufficient and associated with risk of alloimmunization and blood-borne infection. These limitations could be addressed by the generation of autologous megakaryocytes (MKs) derived in vitro from somatic cells with the ability to engraft and differentiate in vivo. Here, we show that overexpression of a defined set of six transcription factors effic
Bit-error rate measurements for ON-OFF keying modulation at multigigabit per second rates over a V-band wireless link are presented. Serial data-rates from 2.5 to 20 Gb/s were studied for a 231-1 bit random sequence. Error-free data transfer over a 0.3-m link was achieved at up to 10 Gb/s. Acceptable bit-error rates, <10-5 and 10-3, were measured at up to 1.5 m for 10- and 15-Gb/s data-rate, respe
Migratory songbirds carry an inherited capacity to migrate several thousand kilometers each year crossing continental landmasses and barriers between distant breeding sites and wintering areas. How individual songbirds manage with extreme precision to find their way is still largely unknown. The functional characteristics of biological compasses used by songbird migrants has mainly been investigat
Type 2 diabetes (T2D) is a complex disease with widespread physiological insults to the regulation of metabolic homeostasis, above all glycaemic regulation. The pathogenesis of T2D and its progression is broadly understood to be through a gradual decrease in peripheral insulin sensitivity, a compensatory rise in insulin secretion, and a gradual decline in beta-cell function, resulting in glycaemic
